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Three-Dimensional In Vitro Staphylococcus aureus Abscess Communities Display Antibiotic Tolerance and Protection from Neutrophil Clearance. / Hofstee, Marloes I.; Riool, Martijn; Terjajevs, Igors et al.

In: Infection and immunity, Vol. 88, No. 11, e00293, 11.2020.

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Hofstee MI, Riool M, Terjajevs I, Thompson K, Stoddart MJ, Richards RG et al. Three-Dimensional In Vitro Staphylococcus aureus Abscess Communities Display Antibiotic Tolerance and Protection from Neutrophil Clearance. Infection and immunity. 2020 Nov;88(11):e00293. doi: 10.1128/IAI.00293-20

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@article{c9a894e82d2546a39965fc78e6d525f9,
title = "Three-Dimensional In Vitro Staphylococcus aureus Abscess Communities Display Antibiotic Tolerance and Protection from Neutrophil Clearance",
abstract = "Staphylococcus aureus is a prominent human pathogen in bone and soft-tissue infections. Pathophysiology involves abscess formation, which consists of central staphylococcal abscess communities (SACs), surrounded by a fibrin pseudocapsule and infiltrating immune cells. Protection against the ingress of immune cells such as neutrophils, or tolerance to antibiotics, remains largely unknown for SACs and is limited by the lack of availability of in vitro models. We describe a three-dimensional in vitro model of SACs grown in a human plasma-supplemented collagen gel. The in vitro SACs reached their maximum size by 24 h and elaborated a fibrin pseudocapsule, as confirmed by electron and immunofluorescence microscopy. The in vitro SACs tolerated 100× the MIC of gentamicin alone and in combination with rifampin, while planktonic controls and mechanically dispersed SACs were efficiently killed. To simulate a host response, SACs were exposed to differentiated PLB-985 neutrophil-like (dPLB) cells and to primary human neutrophils at an early stage of SAC formation or after maturation at 24 h. Both cell types were unable to clear mature in vitro SACs, but dPLB cells prevented SAC growth upon early exposure before pseudocapsule maturation. Neutrophil exposure after plasmin pretreatment of the SACs resulted in a significant decrease in the number of bacteria within the SACs. The in vitro SAC model mimics key in vivo features, offers a new tool to study host-pathogen interactions and drug efficacy assessment, and has revealed the functionality of the S. aureus pseudocapsule in protecting the bacteria from host phagocytic responses and antibiotics.",
keywords = "3-dimensional, Antibiotic tolerance, Bacterium-host cell interactions, Fibrin pseudocapsule, In vitro model, Staphylococcal abscess communities, Staphylococcus aureus",
author = "Hofstee, {Marloes I.} and Martijn Riool and Igors Terjajevs and Keith Thompson and Stoddart, {Martin J.} and Richards, {R. Geoff} and Zaat, {Sebastian A. J.} and Moriarty, {T. Fintan}",
year = "2020",
month = nov,
doi = "10.1128/IAI.00293-20",
language = "English",
volume = "88",
journal = "Infection and immunity",
issn = "0019-9567",
publisher = "American Society for Microbiology",
number = "11",

}

RIS

TY - JOUR

T1 - Three-Dimensional In Vitro Staphylococcus aureus Abscess Communities Display Antibiotic Tolerance and Protection from Neutrophil Clearance

AU - Hofstee, Marloes I.

AU - Riool, Martijn

AU - Terjajevs, Igors

AU - Thompson, Keith

AU - Stoddart, Martin J.

AU - Richards, R. Geoff

AU - Zaat, Sebastian A. J.

AU - Moriarty, T. Fintan

PY - 2020/11

Y1 - 2020/11

N2 - Staphylococcus aureus is a prominent human pathogen in bone and soft-tissue infections. Pathophysiology involves abscess formation, which consists of central staphylococcal abscess communities (SACs), surrounded by a fibrin pseudocapsule and infiltrating immune cells. Protection against the ingress of immune cells such as neutrophils, or tolerance to antibiotics, remains largely unknown for SACs and is limited by the lack of availability of in vitro models. We describe a three-dimensional in vitro model of SACs grown in a human plasma-supplemented collagen gel. The in vitro SACs reached their maximum size by 24 h and elaborated a fibrin pseudocapsule, as confirmed by electron and immunofluorescence microscopy. The in vitro SACs tolerated 100× the MIC of gentamicin alone and in combination with rifampin, while planktonic controls and mechanically dispersed SACs were efficiently killed. To simulate a host response, SACs were exposed to differentiated PLB-985 neutrophil-like (dPLB) cells and to primary human neutrophils at an early stage of SAC formation or after maturation at 24 h. Both cell types were unable to clear mature in vitro SACs, but dPLB cells prevented SAC growth upon early exposure before pseudocapsule maturation. Neutrophil exposure after plasmin pretreatment of the SACs resulted in a significant decrease in the number of bacteria within the SACs. The in vitro SAC model mimics key in vivo features, offers a new tool to study host-pathogen interactions and drug efficacy assessment, and has revealed the functionality of the S. aureus pseudocapsule in protecting the bacteria from host phagocytic responses and antibiotics.

AB - Staphylococcus aureus is a prominent human pathogen in bone and soft-tissue infections. Pathophysiology involves abscess formation, which consists of central staphylococcal abscess communities (SACs), surrounded by a fibrin pseudocapsule and infiltrating immune cells. Protection against the ingress of immune cells such as neutrophils, or tolerance to antibiotics, remains largely unknown for SACs and is limited by the lack of availability of in vitro models. We describe a three-dimensional in vitro model of SACs grown in a human plasma-supplemented collagen gel. The in vitro SACs reached their maximum size by 24 h and elaborated a fibrin pseudocapsule, as confirmed by electron and immunofluorescence microscopy. The in vitro SACs tolerated 100× the MIC of gentamicin alone and in combination with rifampin, while planktonic controls and mechanically dispersed SACs were efficiently killed. To simulate a host response, SACs were exposed to differentiated PLB-985 neutrophil-like (dPLB) cells and to primary human neutrophils at an early stage of SAC formation or after maturation at 24 h. Both cell types were unable to clear mature in vitro SACs, but dPLB cells prevented SAC growth upon early exposure before pseudocapsule maturation. Neutrophil exposure after plasmin pretreatment of the SACs resulted in a significant decrease in the number of bacteria within the SACs. The in vitro SAC model mimics key in vivo features, offers a new tool to study host-pathogen interactions and drug efficacy assessment, and has revealed the functionality of the S. aureus pseudocapsule in protecting the bacteria from host phagocytic responses and antibiotics.

KW - 3-dimensional

KW - Antibiotic tolerance

KW - Bacterium-host cell interactions

KW - Fibrin pseudocapsule

KW - In vitro model

KW - Staphylococcal abscess communities

KW - Staphylococcus aureus

UR - http://www.scopus.com/inward/record.url?scp=85093889948&partnerID=8YFLogxK

U2 - 10.1128/IAI.00293-20

DO - 10.1128/IAI.00293-20

M3 - Article

C2 - 32817328

VL - 88

JO - Infection and immunity

JF - Infection and immunity

SN - 0019-9567

IS - 11

M1 - e00293

ER -

ID: 13974859