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Opportunities and short-comings of the axolotl salamander heart as a model system of human single ventricle and excessive trabeculation. / Meyer, Sophie; Lauridsen, Henrik; Pedersen, Kathrine et al.

In: Scientific reports, Vol. 12, No. 1, 20491, 01.12.2022, p. 20491.

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Meyer S, Lauridsen H, Pedersen K, Andersson SA, van Ooij P, Willems T et al. Opportunities and short-comings of the axolotl salamander heart as a model system of human single ventricle and excessive trabeculation. Scientific reports. 2022 Dec 1;12(1):20491. 20491. doi: 10.1038/s41598-022-24442-9, 10.1038/s41598-022-24442-9

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Meyer, Sophie ; Lauridsen, Henrik ; Pedersen, Kathrine et al. / Opportunities and short-comings of the axolotl salamander heart as a model system of human single ventricle and excessive trabeculation. In: Scientific reports. 2022 ; Vol. 12, No. 1. pp. 20491.

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@article{574e70e79b354365a7ee383c11801ecc,
title = "Opportunities and short-comings of the axolotl salamander heart as a model system of human single ventricle and excessive trabeculation",
abstract = "Few experimental model systems are available for the rare congenital heart diseases of double inlet left ventricle (DILV), a subgroup of univentricular hearts, and excessive trabeculation (ET), or noncompaction. Here, we explore the heart of the axolotl salamander (Ambystoma mexicanum, Shaw 1789) as model system of these diseases. Using micro-echocardiography, we assessed the form and function of the heart of the axolotl, an amphibian, and compared this to human DILV (n = 3). The main finding was that both in the axolotl and DILV, blood flows of disparate oxygen saturation can stay separated in a single ventricle. In the axolotl there is a solitary ventricular inlet and outlet, whereas in DILV there are two separate inlets and outlets. Axolotls had a lower resting heart rate compared to DILV (22 vs. 72 beats per minute), lower ejection fraction (47 vs. 58%), and their oxygen consumption at rest was higher than peak oxygen consumption in DILV (30 vs. 17 ml min−1 kg−1). Concerning the ventricular myocardial organization, histology showed trabeculations in ET (n = 5) are much closer to the normal human setting than to the axolotl setting. We conclude that the axolotl heart resembles some aspects of DILV and ET albeit substantial species differences exist.",
keywords = "Humans, Animals, Ambystoma mexicanum, Univentricular Heart, Urodela, Heart, Cardiovascular Abnormalities",
author = "Sophie Meyer and Henrik Lauridsen and Kathrine Pedersen and Andersson, {Sofie Amalie} and {van Ooij}, Pim and Tineke Willems and Berger, {Rolf M. F.} and Tjark Ebels and Bjarke Jensen",
note = "Publisher Copyright: {\textcopyright} 2022, The Author(s).",
year = "2022",
month = dec,
day = "1",
doi = "10.1038/s41598-022-24442-9",
language = "English",
volume = "12",
pages = "20491",
journal = "Scientific reports",
issn = "2045-2322",
publisher = "Nature Publishing Group",
number = "1",

}

RIS

TY - JOUR

T1 - Opportunities and short-comings of the axolotl salamander heart as a model system of human single ventricle and excessive trabeculation

AU - Meyer, Sophie

AU - Lauridsen, Henrik

AU - Pedersen, Kathrine

AU - Andersson, Sofie Amalie

AU - van Ooij, Pim

AU - Willems, Tineke

AU - Berger, Rolf M. F.

AU - Ebels, Tjark

AU - Jensen, Bjarke

N1 - Publisher Copyright: © 2022, The Author(s).

PY - 2022/12/1

Y1 - 2022/12/1

N2 - Few experimental model systems are available for the rare congenital heart diseases of double inlet left ventricle (DILV), a subgroup of univentricular hearts, and excessive trabeculation (ET), or noncompaction. Here, we explore the heart of the axolotl salamander (Ambystoma mexicanum, Shaw 1789) as model system of these diseases. Using micro-echocardiography, we assessed the form and function of the heart of the axolotl, an amphibian, and compared this to human DILV (n = 3). The main finding was that both in the axolotl and DILV, blood flows of disparate oxygen saturation can stay separated in a single ventricle. In the axolotl there is a solitary ventricular inlet and outlet, whereas in DILV there are two separate inlets and outlets. Axolotls had a lower resting heart rate compared to DILV (22 vs. 72 beats per minute), lower ejection fraction (47 vs. 58%), and their oxygen consumption at rest was higher than peak oxygen consumption in DILV (30 vs. 17 ml min−1 kg−1). Concerning the ventricular myocardial organization, histology showed trabeculations in ET (n = 5) are much closer to the normal human setting than to the axolotl setting. We conclude that the axolotl heart resembles some aspects of DILV and ET albeit substantial species differences exist.

AB - Few experimental model systems are available for the rare congenital heart diseases of double inlet left ventricle (DILV), a subgroup of univentricular hearts, and excessive trabeculation (ET), or noncompaction. Here, we explore the heart of the axolotl salamander (Ambystoma mexicanum, Shaw 1789) as model system of these diseases. Using micro-echocardiography, we assessed the form and function of the heart of the axolotl, an amphibian, and compared this to human DILV (n = 3). The main finding was that both in the axolotl and DILV, blood flows of disparate oxygen saturation can stay separated in a single ventricle. In the axolotl there is a solitary ventricular inlet and outlet, whereas in DILV there are two separate inlets and outlets. Axolotls had a lower resting heart rate compared to DILV (22 vs. 72 beats per minute), lower ejection fraction (47 vs. 58%), and their oxygen consumption at rest was higher than peak oxygen consumption in DILV (30 vs. 17 ml min−1 kg−1). Concerning the ventricular myocardial organization, histology showed trabeculations in ET (n = 5) are much closer to the normal human setting than to the axolotl setting. We conclude that the axolotl heart resembles some aspects of DILV and ET albeit substantial species differences exist.

KW - Humans

KW - Animals

KW - Ambystoma mexicanum

KW - Univentricular Heart

KW - Urodela

KW - Heart

KW - Cardiovascular Abnormalities

UR - http://www.scopus.com/inward/record.url?scp=85142829425&partnerID=8YFLogxK

U2 - 10.1038/s41598-022-24442-9

DO - 10.1038/s41598-022-24442-9

M3 - Article

C2 - 36443330

VL - 12

SP - 20491

JO - Scientific reports

JF - Scientific reports

SN - 2045-2322

IS - 1

M1 - 20491

ER -

ID: 28413181