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Haemodynamic characterisation of different endotypes in coronary artery vasospasm in reaction to acetylcholine. / Feenstra, Rutger G. T.; Boerhout, Coen K. M.; Vink, Caitlin E. M. et al.

In: International journal of cardiology. Heart & vasculature, Vol. 42, 101105, 01.10.2022.

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Feenstra RGT, Boerhout CKM, Vink CEM, Woudstra J, Wittekoek ME, de Waard GA et al. Haemodynamic characterisation of different endotypes in coronary artery vasospasm in reaction to acetylcholine. International journal of cardiology. Heart & vasculature. 2022 Oct 1;42:101105. doi: 10.1016/j.ijcha.2022.101105

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Feenstra, Rutger G. T. ; Boerhout, Coen K. M. ; Vink, Caitlin E. M. et al. / Haemodynamic characterisation of different endotypes in coronary artery vasospasm in reaction to acetylcholine. In: International journal of cardiology. Heart & vasculature. 2022 ; Vol. 42.

BibTeX

@article{3e2fbaee7d47409cb876dbda2b90b04a,
title = "Haemodynamic characterisation of different endotypes in coronary artery vasospasm in reaction to acetylcholine",
abstract = "Background: Vasoreactivity testing with high-dose acetylcholine is considered vasospasm provocation and low-dose as endothelial function testing. Aims: To assess the changes in reaction to low- and high-dose acetylcholine in the endotypes of CAS as defined by the Coronary Vasomotor Disorders International Study Group (COVADIS) working group. Methods: Changes in coronary epicardial diameter, coronary blood flow (CBF) and vascular resistance were determined at low-dose acetylcholine. Results: A total of 88 ANOCA patients were included in this analysis. In the negative group (n = 14) incremental infusion of acetylcholine produced a progressive increase in CBF (p = 0.008). In reaction to low-dose acetylcholine, the epicardial vasospasm group (n = 30) is characterised by epicardial vasoconstriction that is significantly more severe compared to the microvascular vasospasm group (p = 0.004)(n = 23). The equivocal group (n = 21) is characterised by an increase in CBF and reduction in vascular resistance that are both significantly different compared to the epicardial vasospasm group (p = 0.036 and p = 0.007, respectively). High-dose acetylcholine decreased epicardial diameter and CBF significantly in the epicardial vasospasm, microvascular vasospasm and in the equivocal group (all p < 0.05. Vascular resistance increased significantly in the epicardial vasospasm group (p < 0.001) and equivocal group (p = 0.009). Conclusion: In reaction to low-dose acetylcholine the negative and equivocal endotype has haemodynamic changes that suggest intact endothelium. In reaction to high-dose acetylcholine the epicardial vasospasm, microvascular vasospasm and equivocal endotype have hemodynamic changes that suggest VSMC-hyperreactivity. These results suggest that the equivocal endotype is a positive test comparable to microvascular vasospasm in the presence of normal endothelial function.",
keywords = "ANOCA, Acetylcholine, Coronary artery spasm, Haemodynamic changes",
author = "Feenstra, {Rutger G. T.} and Boerhout, {Coen K. M.} and Vink, {Caitlin E. M.} and Janneke Woudstra and Wittekoek, {Marianne E.} and {de Waard}, {Guus A.} and Yolande Appelman and Eringa, {Etto C.} and Marques, {Koen M. J.} and {de Winter}, {Robbert J.} and {van de Hoef}, {Tim P.} and Beijk, {Marcel A. M.} and Piek, {Jan J.}",
note = "Publisher Copyright: {\textcopyright} 2022",
year = "2022",
month = oct,
day = "1",
doi = "10.1016/j.ijcha.2022.101105",
language = "English",
volume = "42",
journal = "International journal of cardiology. Heart & vasculature",
issn = "2352-9067",
publisher = "Elsevier BV",

}

RIS

TY - JOUR

T1 - Haemodynamic characterisation of different endotypes in coronary artery vasospasm in reaction to acetylcholine

AU - Feenstra, Rutger G. T.

AU - Boerhout, Coen K. M.

AU - Vink, Caitlin E. M.

AU - Woudstra, Janneke

AU - Wittekoek, Marianne E.

AU - de Waard, Guus A.

AU - Appelman, Yolande

AU - Eringa, Etto C.

AU - Marques, Koen M. J.

AU - de Winter, Robbert J.

AU - van de Hoef, Tim P.

AU - Beijk, Marcel A. M.

AU - Piek, Jan J.

N1 - Publisher Copyright: © 2022

PY - 2022/10/1

Y1 - 2022/10/1

N2 - Background: Vasoreactivity testing with high-dose acetylcholine is considered vasospasm provocation and low-dose as endothelial function testing. Aims: To assess the changes in reaction to low- and high-dose acetylcholine in the endotypes of CAS as defined by the Coronary Vasomotor Disorders International Study Group (COVADIS) working group. Methods: Changes in coronary epicardial diameter, coronary blood flow (CBF) and vascular resistance were determined at low-dose acetylcholine. Results: A total of 88 ANOCA patients were included in this analysis. In the negative group (n = 14) incremental infusion of acetylcholine produced a progressive increase in CBF (p = 0.008). In reaction to low-dose acetylcholine, the epicardial vasospasm group (n = 30) is characterised by epicardial vasoconstriction that is significantly more severe compared to the microvascular vasospasm group (p = 0.004)(n = 23). The equivocal group (n = 21) is characterised by an increase in CBF and reduction in vascular resistance that are both significantly different compared to the epicardial vasospasm group (p = 0.036 and p = 0.007, respectively). High-dose acetylcholine decreased epicardial diameter and CBF significantly in the epicardial vasospasm, microvascular vasospasm and in the equivocal group (all p < 0.05. Vascular resistance increased significantly in the epicardial vasospasm group (p < 0.001) and equivocal group (p = 0.009). Conclusion: In reaction to low-dose acetylcholine the negative and equivocal endotype has haemodynamic changes that suggest intact endothelium. In reaction to high-dose acetylcholine the epicardial vasospasm, microvascular vasospasm and equivocal endotype have hemodynamic changes that suggest VSMC-hyperreactivity. These results suggest that the equivocal endotype is a positive test comparable to microvascular vasospasm in the presence of normal endothelial function.

AB - Background: Vasoreactivity testing with high-dose acetylcholine is considered vasospasm provocation and low-dose as endothelial function testing. Aims: To assess the changes in reaction to low- and high-dose acetylcholine in the endotypes of CAS as defined by the Coronary Vasomotor Disorders International Study Group (COVADIS) working group. Methods: Changes in coronary epicardial diameter, coronary blood flow (CBF) and vascular resistance were determined at low-dose acetylcholine. Results: A total of 88 ANOCA patients were included in this analysis. In the negative group (n = 14) incremental infusion of acetylcholine produced a progressive increase in CBF (p = 0.008). In reaction to low-dose acetylcholine, the epicardial vasospasm group (n = 30) is characterised by epicardial vasoconstriction that is significantly more severe compared to the microvascular vasospasm group (p = 0.004)(n = 23). The equivocal group (n = 21) is characterised by an increase in CBF and reduction in vascular resistance that are both significantly different compared to the epicardial vasospasm group (p = 0.036 and p = 0.007, respectively). High-dose acetylcholine decreased epicardial diameter and CBF significantly in the epicardial vasospasm, microvascular vasospasm and in the equivocal group (all p < 0.05. Vascular resistance increased significantly in the epicardial vasospasm group (p < 0.001) and equivocal group (p = 0.009). Conclusion: In reaction to low-dose acetylcholine the negative and equivocal endotype has haemodynamic changes that suggest intact endothelium. In reaction to high-dose acetylcholine the epicardial vasospasm, microvascular vasospasm and equivocal endotype have hemodynamic changes that suggest VSMC-hyperreactivity. These results suggest that the equivocal endotype is a positive test comparable to microvascular vasospasm in the presence of normal endothelial function.

KW - ANOCA

KW - Acetylcholine

KW - Coronary artery spasm

KW - Haemodynamic changes

UR - http://www.scopus.com/inward/record.url?scp=85135914028&partnerID=8YFLogxK

U2 - 10.1016/j.ijcha.2022.101105

DO - 10.1016/j.ijcha.2022.101105

M3 - Article

C2 - 36017267

VL - 42

JO - International journal of cardiology. Heart & vasculature

JF - International journal of cardiology. Heart & vasculature

SN - 2352-9067

M1 - 101105

ER -

ID: 25732884