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Fluid overload due to intravenous fluid therapy for vaso-occlusive crisis in sickle cell disease: incidence and risk factors. / Gaartman, Aafke E.; Sayedi, Ajab K.; Gerritsma, Jorn J. et al.

In: British journal of haematology, Vol. 194, No. 5, 01.09.2021, p. 899-907.

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@article{21cda550e49a46ecb55386a8a7e070d0,
title = "Fluid overload due to intravenous fluid therapy for vaso-occlusive crisis in sickle cell disease: incidence and risk factors",
abstract = "Intravenous fluid therapy (IV-FT) is routinely used in the treatment of vaso-occlusive crises (VOCs), as dehydration possibly promotes and sustains erythrocyte sickling. Patients with sickle cell disease (SCD) are at risk of developing diastolic dysfunction and fluid overload due to IV-FT. However, data on the adverse effects of IV-FT for VOC is sparse. We aimed to evaluate the incidence and risk factors of fluid overload due to IV-FT in patients with SCD. Consecutive hospitalisations for VOC treated with IV-FT between September 2016 and September 2018 were retrospectively analysed. The median (interquartile range) age was 25·0 (18·3–33·8) years and 65% had a severe genotype (HbSS/HbSβ0-thal). Fluid overload occurred in 21% of 100 patients. Hospital stay was longer in patients with fluid overload (6·0 vs. 4·0 days, P = 0·037). A positive history of fluid overload (P = 0·017), lactate dehydrogenase level (P = 0·011), and top-up transfusion during admission (P = 0·005) were independently associated with fluid overload occurrence. IV-FT was not reduced in 86% of patients despite a previous history of fluid overload. Fluid overload is frequently encountered during IV-FT for VOC. IV-FT is often not adjusted despite a positive history of fluid overload or when top-up transfusion is indicated, emphasising the need for more awareness of this complication and a personalised approach.",
keywords = "fluid overload, fluid therapy, pulmonary oedema, sickle cell disease, vaso-occlusive crisis",
author = "Gaartman, {Aafke E.} and Sayedi, {Ajab K.} and Gerritsma, {Jorn J.} and {de Back}, {Tim R.} and {van Tuijn}, {Charlotte F.} and Tang, {Man Wai} and Harri{\"e}t Heijboer and {de Heer}, Koen and Biemond, {Bart J.} and Erfan Nur",
note = "Funding Information: E.J. van Beers: Van Creveldkliniek, University Medical Center Utrecht, Heidelberglaan 100, 3584 CX Utrecht, the Netherlands; B.J. Biemond: Department of Hematology, Amsterdam UMC, University of Amsterdam, Meibergdreef 9, 1105 AZ, Amsterdam, the Netherlands; M. Beijlevelt: Department of Pediatric Hematology, Emma Children?s Hospital, Amsterdam UMC, University of Amsterdam, Meibergdreef 9, Amsterdam, Netherlands; P.P.T. Brons: Department of Pediatric Hematology, Amalia Children's Hospital, Radboudumc, University Medical Center, Geert Grooteplein Zuid 10, 6500 HB, Nijmegen, the Netherlands; M.H. Cnossen: Department of Pediatric Hematology, Erasmus University Medical Center?Sophia Children's Hospital, Dr. Molewaterplein 40, 3015 GD Rotterdam, the Netherlands; K. Fijnvandraat: Department of Pediatric Hematology, Emma Children?s Hospital, Amsterdam UMC, University of Amsterdam, Meibergdreef 9, Amsterdam, Netherlands; Sanquin Research, Department of Molecular Cellular Hemostasis, Sanquin Research, PO Box?9190, 1006 AD Amsterdam, the Netherlands; H. Heijboer: Department of Pediatric Hematology, Emma Children?s Hospital, Amsterdam UMC, University of Amsterdam, Meibergdreef 9, Amsterdam, Netherlands; F. Hofstede: Department of Hematology, Haga Hospital, Els Borst-Eilersplein 275, 2545 AA Den Haag, the Netherlands; J.L.H. Kerkhoffs: Department of Hematology, Haga Hospital, Els Borst-Eilersplein 275, 2545 AA Den Haag, the Netherlands; E. Nur: Department of Hematology, Amsterdam UMC, University of Amsterdam, Meibergdreef 9, 1105 AZ, Amsterdam, the Netherlands; C. Ootjers: Department of Hematology, Leiden University Medical Center, Albinusdreef 2, 2333 ZA Leiden, the Netherlands; P.J. de Pagter: Department of Pediatric Hematology, Erasmus University Medical Center ? Sophia Children's Hospital,?Dr. Molewaterplein 40, 3015 GD Rotterdam, the Netherlands; Department of Pediatric Hematology, Willem-Alexander Children's Hospital, Leiden University Medical Center, Albinusdreef 2, 2333 ZA Leiden, the Netherlands; A.W. Rijneveld: Department of Hematology, Erasmus University Medical Center, Dr. Molewaterplein 40, 3015 GD Rotterdam, the Netherlands; S.E.M. Schols: Department of Hematology, Radboud University Medical Center, Geert Grooteplein Zuid 10, 6525 GA, Nijmegen, the Netherland; F.J. Smiers: Department of Pediatric Hematology, Willem-Alexander Children's Hospital, Leiden University Medical Center, Albinusdreef 2, 2333 ZA Leiden, the Netherlands; M.H. Suiker: Van Creveldkliniek, University Medical Center Utrecht, Heidelberglaan 100, 3584 CX Utrecht, the Netherlands; C.F.J. van Tuijn: Department of Hematology, Amsterdam UMC, University of Amsterdam, Meibergdreef 9, 1105 AZ, Amsterdam, the Netherlands; I. van Vliet: Department of Pediatric Hematology, Erasmus University Medical Center, Sophia Children's Hospital, Dr. Molewaterplein 40, 3015 GD Rotterdam, the Netherlands; E. Zwagemaker: Van Creveldkliniek, University Medical Center Utrecht, Heidelberglaan 100, 3584 CX Utrecht, the Netherlands. Publisher Copyright: {\textcopyright} 2021 The Authors. British Journal of Haematology published by British Society for Haematology and John Wiley & Sons Ltd",
year = "2021",
month = sep,
day = "1",
doi = "10.1111/bjh.17696",
language = "English",
volume = "194",
pages = "899--907",
journal = "British journal of haematology",
issn = "0007-1048",
publisher = "Wiley-Blackwell",
number = "5",

}

RIS

TY - JOUR

T1 - Fluid overload due to intravenous fluid therapy for vaso-occlusive crisis in sickle cell disease: incidence and risk factors

AU - Gaartman, Aafke E.

AU - Sayedi, Ajab K.

AU - Gerritsma, Jorn J.

AU - de Back, Tim R.

AU - van Tuijn, Charlotte F.

AU - Tang, Man Wai

AU - Heijboer, Harriët

AU - de Heer, Koen

AU - Biemond, Bart J.

AU - Nur, Erfan

N1 - Funding Information: E.J. van Beers: Van Creveldkliniek, University Medical Center Utrecht, Heidelberglaan 100, 3584 CX Utrecht, the Netherlands; B.J. Biemond: Department of Hematology, Amsterdam UMC, University of Amsterdam, Meibergdreef 9, 1105 AZ, Amsterdam, the Netherlands; M. Beijlevelt: Department of Pediatric Hematology, Emma Children?s Hospital, Amsterdam UMC, University of Amsterdam, Meibergdreef 9, Amsterdam, Netherlands; P.P.T. Brons: Department of Pediatric Hematology, Amalia Children's Hospital, Radboudumc, University Medical Center, Geert Grooteplein Zuid 10, 6500 HB, Nijmegen, the Netherlands; M.H. Cnossen: Department of Pediatric Hematology, Erasmus University Medical Center?Sophia Children's Hospital, Dr. Molewaterplein 40, 3015 GD Rotterdam, the Netherlands; K. Fijnvandraat: Department of Pediatric Hematology, Emma Children?s Hospital, Amsterdam UMC, University of Amsterdam, Meibergdreef 9, Amsterdam, Netherlands; Sanquin Research, Department of Molecular Cellular Hemostasis, Sanquin Research, PO Box?9190, 1006 AD Amsterdam, the Netherlands; H. Heijboer: Department of Pediatric Hematology, Emma Children?s Hospital, Amsterdam UMC, University of Amsterdam, Meibergdreef 9, Amsterdam, Netherlands; F. Hofstede: Department of Hematology, Haga Hospital, Els Borst-Eilersplein 275, 2545 AA Den Haag, the Netherlands; J.L.H. Kerkhoffs: Department of Hematology, Haga Hospital, Els Borst-Eilersplein 275, 2545 AA Den Haag, the Netherlands; E. Nur: Department of Hematology, Amsterdam UMC, University of Amsterdam, Meibergdreef 9, 1105 AZ, Amsterdam, the Netherlands; C. Ootjers: Department of Hematology, Leiden University Medical Center, Albinusdreef 2, 2333 ZA Leiden, the Netherlands; P.J. de Pagter: Department of Pediatric Hematology, Erasmus University Medical Center ? Sophia Children's Hospital,?Dr. Molewaterplein 40, 3015 GD Rotterdam, the Netherlands; Department of Pediatric Hematology, Willem-Alexander Children's Hospital, Leiden University Medical Center, Albinusdreef 2, 2333 ZA Leiden, the Netherlands; A.W. Rijneveld: Department of Hematology, Erasmus University Medical Center, Dr. Molewaterplein 40, 3015 GD Rotterdam, the Netherlands; S.E.M. Schols: Department of Hematology, Radboud University Medical Center, Geert Grooteplein Zuid 10, 6525 GA, Nijmegen, the Netherland; F.J. Smiers: Department of Pediatric Hematology, Willem-Alexander Children's Hospital, Leiden University Medical Center, Albinusdreef 2, 2333 ZA Leiden, the Netherlands; M.H. Suiker: Van Creveldkliniek, University Medical Center Utrecht, Heidelberglaan 100, 3584 CX Utrecht, the Netherlands; C.F.J. van Tuijn: Department of Hematology, Amsterdam UMC, University of Amsterdam, Meibergdreef 9, 1105 AZ, Amsterdam, the Netherlands; I. van Vliet: Department of Pediatric Hematology, Erasmus University Medical Center, Sophia Children's Hospital, Dr. Molewaterplein 40, 3015 GD Rotterdam, the Netherlands; E. Zwagemaker: Van Creveldkliniek, University Medical Center Utrecht, Heidelberglaan 100, 3584 CX Utrecht, the Netherlands. Publisher Copyright: © 2021 The Authors. British Journal of Haematology published by British Society for Haematology and John Wiley & Sons Ltd

PY - 2021/9/1

Y1 - 2021/9/1

N2 - Intravenous fluid therapy (IV-FT) is routinely used in the treatment of vaso-occlusive crises (VOCs), as dehydration possibly promotes and sustains erythrocyte sickling. Patients with sickle cell disease (SCD) are at risk of developing diastolic dysfunction and fluid overload due to IV-FT. However, data on the adverse effects of IV-FT for VOC is sparse. We aimed to evaluate the incidence and risk factors of fluid overload due to IV-FT in patients with SCD. Consecutive hospitalisations for VOC treated with IV-FT between September 2016 and September 2018 were retrospectively analysed. The median (interquartile range) age was 25·0 (18·3–33·8) years and 65% had a severe genotype (HbSS/HbSβ0-thal). Fluid overload occurred in 21% of 100 patients. Hospital stay was longer in patients with fluid overload (6·0 vs. 4·0 days, P = 0·037). A positive history of fluid overload (P = 0·017), lactate dehydrogenase level (P = 0·011), and top-up transfusion during admission (P = 0·005) were independently associated with fluid overload occurrence. IV-FT was not reduced in 86% of patients despite a previous history of fluid overload. Fluid overload is frequently encountered during IV-FT for VOC. IV-FT is often not adjusted despite a positive history of fluid overload or when top-up transfusion is indicated, emphasising the need for more awareness of this complication and a personalised approach.

AB - Intravenous fluid therapy (IV-FT) is routinely used in the treatment of vaso-occlusive crises (VOCs), as dehydration possibly promotes and sustains erythrocyte sickling. Patients with sickle cell disease (SCD) are at risk of developing diastolic dysfunction and fluid overload due to IV-FT. However, data on the adverse effects of IV-FT for VOC is sparse. We aimed to evaluate the incidence and risk factors of fluid overload due to IV-FT in patients with SCD. Consecutive hospitalisations for VOC treated with IV-FT between September 2016 and September 2018 were retrospectively analysed. The median (interquartile range) age was 25·0 (18·3–33·8) years and 65% had a severe genotype (HbSS/HbSβ0-thal). Fluid overload occurred in 21% of 100 patients. Hospital stay was longer in patients with fluid overload (6·0 vs. 4·0 days, P = 0·037). A positive history of fluid overload (P = 0·017), lactate dehydrogenase level (P = 0·011), and top-up transfusion during admission (P = 0·005) were independently associated with fluid overload occurrence. IV-FT was not reduced in 86% of patients despite a previous history of fluid overload. Fluid overload is frequently encountered during IV-FT for VOC. IV-FT is often not adjusted despite a positive history of fluid overload or when top-up transfusion is indicated, emphasising the need for more awareness of this complication and a personalised approach.

KW - fluid overload

KW - fluid therapy

KW - pulmonary oedema

KW - sickle cell disease

KW - vaso-occlusive crisis

UR - http://www.scopus.com/inward/record.url?scp=85110181694&partnerID=8YFLogxK

U2 - 10.1111/bjh.17696

DO - 10.1111/bjh.17696

M3 - Article

C2 - 34263922

VL - 194

SP - 899

EP - 907

JO - British journal of haematology

JF - British journal of haematology

SN - 0007-1048

IS - 5

ER -

ID: 19613539