Research interests

 My research aims at understanding why an overloaded heart ultimately fails. Our leading thought is that the transition towards failure is governed by regulatory mechanisms that cause an accumulation of maladaptive changes. Our current focus is on genetic mechanisms and mechanisms involving RNA regulatory molecuales like microRNAs, lncRNAs and RNA splicing as key regulators of the adapative and maladaptive changes in the overloaded heart.

Research output

  1. Seeing the disease before it has manifested itself

    Research output: Contribution to journalEditorialAcademicpeer-review

  2. Biomarkers to diagnose ventricular dysfunction in childhood cancer survivors: A systematic review

    Research output: Contribution to journalArticleAcademicpeer-review

  3. Circular RNAs open a new chapter in cardiovascular biology

    Research output: Contribution to journalReview articleAcademicpeer-review

View all (237) »

ID: 78422